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Edited by Burton H. Singer, University of Florida, Gainesville, FL, and approved November 10, 2017 (received for review August 23, 2017)
Article Figures & SI
Figures
- Fig. 1.
(A) Average prevalence of HPV types in the study population, with SEs calculated across visits. (B) Prevalence over time of the six types analyzed.
- Fig. 2.
Dynamics for one individual host i and one HPV type j under the additional risk model (Eq. 4). S and I denote susceptible and infected, respectively. The duration of each infection is drawn from a gamma distribution with mean
- Fig. 3.
(A) Log estimate of the additional risk d by sexual subclass for each HPV type [maximum likelihood estimate (MLE) and 95% confidence interval]. (B) Predicted number of unique individuals infected with HPV16 during follow-up across 1,000 simulations under the additional risk model compared with the memoryless model and the data.
- Fig. 4.
Inferred effect of host-specific covariates (maximum likelihood parameter estimate and the 95% confidence interval). Colored cells denote statistically significant positive (blue) and negative (red) effects.
Data supplements
Supporting Information
High HPV prevalence arises by recurring infection
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