Significance

Mitogen-activated protein kinases (MAPKs) function in all eukaryotes in signaling extracellular stimuli to intracellular responses and ultimately link them to chromatin events by targeting transcription factors and chromatin remodeling complexes. In plants, MAPKs play crucial roles in immunity, development, and stress responses, but so far no attempts have been made to identify phosphorylation of chromatin-associated proteins. By using a phosphoproteomic approach on MAPK mutants, we identified a number of chromatin-associated MAPK substrates and characterize an AT-hook motif containing nuclear localized (AHL) DNA-binding protein 13 in plant immunity and demonstrate that phosphorylation regulates AHL13 protein stability and, in turn, its function in response to pathogens.

Abstract

In many eukaryotic systems during immune responses, mitogen-activated protein kinases (MAPKs) link cytoplasmic signaling to chromatin events by targeting transcription factors, chromatin remodeling complexes, and the RNA polymerase machinery. So far, knowledge on these events is scarce in plants and no attempts have been made to focus on phosphorylation events of chromatin-associated proteins. Here we carried out chromatin phosphoproteomics upon elicitor-induced activation of Arabidopsis. The events in WT were compared with those in mpk3, mpk4, and mpk6 mutant plants to decipher specific MAPK targets. Our study highlights distinct signaling networks involving MPK3, MPK4, and MPK6 in chromatin organization and modification, as well as in RNA transcription and processing. Among the chromatin targets, we characterized the AT-hook motif containing nuclear localized (AHL) DNA-binding protein AHL13 as a substrate of immune MAPKs. AHL13 knockout mutant plants are compromised in pathogen-associated molecular pattern (PAMP)-induced reactive oxygen species production, expression of defense genes, and PAMP-triggered immunity. Transcriptome analysis revealed that AHL13 regulates key factors of jasmonic acid biosynthesis and signaling and affects immunity toward Pseudomonas syringae and Botrytis cinerea pathogens. Mutational analysis of the phosphorylation sites of AHL13 demonstrated that phosphorylation regulates AHL13 protein stability and thereby its immune functions.

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Data Availability

The MS proteomics data have been deposited to the ProteomeXchange Consortium via PRIDE (91) partner repository (dataset identifier PXD009823 and DOI: https://doi.org/10.6019/PXD009823). The RNA-seq data have been deposited in the National Center for Biotechnology Information’s Gene Expression Omnibus database, https://www.ncbi.nlm.nih.gov/geo (accession no. GSE119465).

Acknowledgments

We thank Véronique Legros, Huma Khurram, and Huoming Zhang for technical assistance in mass spectometry; Stéphanie Pateyron and Alexandra Avon for technical support in molecular biology; and Jean Colcombet for kindly providing the constitutively active MAPK vectors. This work was supported by Agence Nationale de la Recherche ANR-2010-JCJC-1608 and ANR-14-CE19-0014 (to D.P.); Investissement d’Avenir Infrastructures Nationales en Biologie et Santé program (ProFI project, ANR-10-INBS-08); and by King Abdullah University of Science and Technology BAS/1/1062-01-01 (to H.H.). The Institute of Plant Sciences Paris-Saclay benefits from the support of the LabEx Saclay Plant Sciences (ANR-10-LABX-0040-SPS). M.A.-T. was supported by a Humboldt-Capes fellowship.

Supporting Information

Appendix (PDF)
Dataset_S01 (XLSX)
Dataset_S02 (XLSX)
Dataset_S03 (XLSX)
Dataset_S04 (XLSX)
Dataset_S05 (XLSX)
Dataset_S06 (XLSX)
Dataset_S07 (XLSX)
Dataset_S08 (XLSX)

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Information & Authors

Information

Published in

The cover image for PNAS Vol.118; No.3
Proceedings of the National Academy of Sciences
Vol. 118 | No. 3
January 19, 2021
PubMed: 33419940

Classifications

Data Availability

The MS proteomics data have been deposited to the ProteomeXchange Consortium via PRIDE (91) partner repository (dataset identifier PXD009823 and DOI: https://doi.org/10.6019/PXD009823). The RNA-seq data have been deposited in the National Center for Biotechnology Information’s Gene Expression Omnibus database, https://www.ncbi.nlm.nih.gov/geo (accession no. GSE119465).

Submission history

Published online: January 8, 2021
Published in issue: January 19, 2021

Keywords

  1. phosphoproteomics
  2. MAPK signaling
  3. chromatin
  4. immunity
  5. AT-hook motif transcription factor

Acknowledgments

We thank Véronique Legros, Huma Khurram, and Huoming Zhang for technical assistance in mass spectometry; Stéphanie Pateyron and Alexandra Avon for technical support in molecular biology; and Jean Colcombet for kindly providing the constitutively active MAPK vectors. This work was supported by Agence Nationale de la Recherche ANR-2010-JCJC-1608 and ANR-14-CE19-0014 (to D.P.); Investissement d’Avenir Infrastructures Nationales en Biologie et Santé program (ProFI project, ANR-10-INBS-08); and by King Abdullah University of Science and Technology BAS/1/1062-01-01 (to H.H.). The Institute of Plant Sciences Paris-Saclay benefits from the support of the LabEx Saclay Plant Sciences (ANR-10-LABX-0040-SPS). M.A.-T. was supported by a Humboldt-Capes fellowship.

Notes

This article is a PNAS Direct Submission. C.Z. is a guest editor invited by the Editorial Board.

Authors

Affiliations

Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia;
Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia;
Centre for Research in Agricultural Genomics, Consejo Superior de Investigaciones Cientificas, Catalan Institute for Food and Agricultural Research and Technology, Autonomous University of Barcelona, University of Barcelona, 08007 Barcelona, Spain;
Department of Biochemistry, Faculty of Science, King Abdulaziz University, 21551 Jeddah, Saudi Arabia;
CNRS, Institut National de Recherche pour l’Agriculture, l’Alimentation, et l’Environnement, University Evry, Institute of Plant Sciences Paris-Saclay, Université Paris-Saclay, 91405 Orsay, France;
Institut National de Recherche pour l’Agriculture, l’Alimentation, et l’Environnement, Institute of Plant Sciences Paris-Saclay, Université de Paris, CNRS, 91405 Orsay, France;
Department of Biological Sciences, College of Sciences and Arts-Rabigh Campus, King Abdulaziz University, 21589 Jeddah, Saudi Arabia;
Plant Immunity Research Center, Seoul National University, 08826 Seoul, Korea;
Kiruthiga Gayathri Mariappan https://orcid.org/0000-0002-0831-8756
Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia;
Marilia Almeida-Trapp
Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia;
Research Group Plant Defense Physiology, Max Planck Institute for Chemical Ecology, 07745 Jena, Germany;
Maria Schlöffel
Center for Plant Molecular Biology, Eberhard-Karls-Universität Tübingen, 72074 Tübingen, Germany;
Emmanuelle Lastrucci
Commissariat à l’Energie Atomique, INSERM, Interdisciplinary Research Institute Grenoble, Large Scale Biology Laboratory, University Grenoble Alpes, 38000 Grenoble, France;
Institut National de Recherche pour l’Agriculture, l’Alimentation, et l’Environnement, Laboratoire de Génétique, Diversité, Ecophysiologie des Céréales, Université Clermont Auvergne, 63000 Clermont Ferrand, France;
Center for Plant Molecular Biology, Eberhard-Karls-Universität Tübingen, 72074 Tübingen, Germany;
Research Group Plant Defense Physiology, Max Planck Institute for Chemical Ecology, 07745 Jena, Germany;
Computational Bioscience Research Center, Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955-6900, Saudi Arabia;
Commissariat à l’Energie Atomique, INSERM, Interdisciplinary Research Institute Grenoble, Large Scale Biology Laboratory, University Grenoble Alpes, 38000 Grenoble, France;
Interdisciplinary Research Institute of Grenoble, Large Scale Biology Laboratory, CNRS, F-38000 Grenoble, France;
CNRS, LAMBE UMR 8587, Université d’Evry Val d’Essonne, 91000 Evry, France;
Division of Biological and Environmental Sciences and Engineering, King Abdullah University of Science and Technology, Thuwal 23955, Saudi Arabia;
Max Perutz Laboratories, University of Vienna, 1030 Vienna, Austria

Notes

2
To whom correspondence may be addressed. Email: [email protected], [email protected], or [email protected].
Author contributions: N.R., J.B., D.P., and H.H. designed research; N.R., M.J., H.M.A., J.B., A.A.A., R.V., M.A.-T., M.S., and D.P. performed research; N.R., J.B., K.G.M., E.L., L.B., A.A.G., A.M., S.T.A., D.P., and H.H. analyzed data; and N.R., H.M.A., J.B., S.T.A., D.P., and H.H. wrote the paper.
1
N.R., M.J., H.M.A., and J.B. contributed equally to this work.

Competing Interests

The authors declare no competing interest.

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    Chromatin phosphoproteomics unravels a function for AT-hook motif nuclear localized protein AHL13 in PAMP-triggered immunity
    Proceedings of the National Academy of Sciences
    • Vol. 118
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